Originally Posted by
edpmedic
"If A then do B and check for outcome," huh? Your what's commonly referred to as a cookbook medic. Not every pt fits into a neat little textbook presentation. For example, consider that many CHF pts developed that CHF secondary to their COPD (emphysema) over the years. Pulmonary HTN is a common contributing factor. They have dyspnea, and they're fairly tight, too tight to hear rales. Do you think they're having a COPD exacerbation? Are they developing APE? Is it both? What do you do first? How do you manage both conditions, and how do you go about that? That requires using at least two protocols. Take a good look at Wake Co, NC's EMS clinical guidelines. Their medics use guidelines, as in best judgment, rather than the simplistic "see A, do B."
Do you understand the mechanism with which succinylcholine (one of the RSI meds) can cause hyperkalemia, and how that can lead to malignant hyperthermia? I'll bet the in house "pharmacology for EMS" that waters down a college pharm course into a week, and only covers the thirty meds or so didn't teach you that.
Do you understand how to use the ETCO2 capnoline (nasal ETCO2 for non-intubated pts) for applications other than verifying tube placement? I'm guessing that the two week watered down "A&P" for EMS that the medic mill gives in lieu of requiring college A&P didn't give you the education to fully understand capnography and capnometry. Can you tell me how to diagnose a STEMI with a pt that has a LBBB or paced rhythm?
To take NVCC's EMS AAS program as an example, you get human biology (A&P), general pharmacology, pathophysiology, advanced patho, a class dedicated to just 12 leads. EMS management is what you study when you progress past the AAS and go for an EMS Bachelors, BTW.
When over 90% of pts in EMS are non-acute, or non-time sensitive, I wouldn't expect the numbers to differ much from the degree medics to non degree medics. We're talking about maybe 5-10% of the pt population that would fare worse if they weren't given more than an O2 NRB and txp. Part of that 10% would be cardiac arrests, who typically stay dead regardless. That opens a whole other can of worms regarding the importance of ALS response and pt outcomes. Consider that a dept's current protocols reflect the medic's lack of education. That's why your friend in Australia, who is likely an Advanced Care Paramedic, which is above the Primary Care Paramedic, can treat and release. In NYC, you can't even give an albuterol in-line neb for an APE pt. Even if you could, that would be jumping protocols, and you would have to spend a few minutes on the phone with the doc-in-the-box giving a complete head to toe while your pt deteriorates.